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By Delbert M. Shankel (auth.), Delbert M. Shankel, Philip E. Hartman, Tsuneo Kada, Alexander Hollaender, Claire M. Wilson, Gregory Kuny (eds.)

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188. 189. 190. 191. 192. 193. 194. 195. 196. 197. 198. , T. Matsumura, N. Sato, T. Miyamoto, T. Ooka, T. Kamada, and H. Abe (1981) Lipid peroxidation in alcoholic liver disease in humans. Alcoholism: Clin. Exp. Res. 5:427-430. , and M. Nagao (1982) The use of mutagenicity to evaluate carcinogenic hazards in our daily lives. A. Heddle, ed. Academic Press, New York, pp. 73-88. , and S. Sato (1983) Mutagens-carcinogens in foods. Cancer Res. ):2415s-2421s. 1. smutase, longevity and specific metabolic-rate.

This shows the uncertainty of whether a substance carcinogenic to one (or even both) of these species is of any carcinogenic risk to humans. Conversely, important human carcinogens may be negative in rats and mice, as was for a long time the case for tobacco and alcohol, the two largest reliably known causes of neoplastic death in the United States. Quantitative Uncertainties In addition to these difficulties with qualitative correspondence, there are major problems with quantitative extrapolations to humans (54,89, 22 B.

Toxicol. 1:2-3. Farber, E. A. Clowes Memorial Lecture. Cancer Res. 44:5463-5474. , S. Parker, and M. Gruenstein (1976) The resistance of putative premalignant liver-cell populations, hyperplastic nodules, to the acute cytotoxic effects of some heptocarcinogens. Cancer Res. 36: 3879-3887. , A. A. Woutersen (1982) Respiratory tract tumours in hamsters exposed to acetaldehyde vapour alone or simultaneously to benzo(a)pyrene or diethylnitrosamine. Eur. J. Canc. Clin. Oncol. 18:13-31. , R. Fulceri, A.

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